Angiotensin AT1 receptor antagonism and protection against cardiovascular end-organ damage

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Angiotensin II receptor blockade and end-organ protection: pharmacological rationale and evidence.

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Angiotensin II (Ang II) and its major heterotrimeric G protein-coupled receptor (GPCR) AT1 have long been believed to mediate end organ damage associated with cardiovascular and metabolic diseases. In the past decade, efforts have been made to dissect intracellular signal transduction mechanisms by which Ang II exerts its pathophysiological effects on target cells and tissues. Recent accumulati...

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Blood pressure and body fluid volume are mostly regulated by the renin-angiotensin system (RAS). It is evident that overactive RAS play a central role in the development of hypertension (HTN) and end organ damage (EOD) associated with HTN. It also plays a key role in a number of pathophysiological mechanisms leading to major cardiovascular (CV) events, such as myocardial infarction (MI), stroke...

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ژورنال

عنوان ژورنال: Journal of Human Hypertension

سال: 1998

ISSN: 0950-9240,1476-5527

DOI: 10.1038/sj.jhh.1000634